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This finding may be compatible with the previous reports indicating that ERGO modulates oxidative damage of the kidney in rats and that decreased ERGO levels may contribute to chronic kidney disease progression implying beneficial roles of ERGO in the kidney [80, 81]

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Deficiency of neuronal p38alpha MAPK attenuates amyloid pathology in Alzheimer disease mouse and cell models through facilitating lysosomal degradation of BACE1

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Experimental and therapeutic medicine

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The results of the clinical studies included in this review highlighted that ALA is a molecule capable of preventing or slowing down various conditions linked to the progression of various diseases, such as neurodegenerative diseases, diabetes, and myocardial and cerebral ischemiareperfusion injury

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